{{Rsnum
|rsid=10401969
|Gene=SUGP1
|Chromosome=19
|position=19296909
|Orientation=plus
|GMAF=0.1065
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
|Gene_s=SUGP1
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 0.9 | 17.7 | 81.4
| HCB | 1.5 | 15.3 | 83.2
| JPT | 0.9 | 15.0 | 84.1
| YRI | 2.7 | 21.8 | 75.5
| ASW | 1.8 | 24.6 | 73.7
| CHB | 1.5 | 15.3 | 83.2
| CHD | 0.9 | 18.3 | 80.7
| GIH | 1.0 | 22.8 | 76.2
| LWK | 8.2 | 30.9 | 60.9
| MEX | 0.0 | 12.1 | 87.9
| MKK | 5.1 | 30.1 | 64.7
| TSI | 0.0 | 7.8 | 92.2
| HapMapRevision=28
}}
{{PMID Auto GWAS
|PMID=19060906
|Trait=LDL cholesterol
|Title=Common variants at 30 loci contribute to polygenic dyslipidemia
|RiskAllele=C
|Pval=2E-8
|OR=0.05
|ORtxt=[-0.03-0.13] SD decrease
|OA=1
}}

{{PharmGKB
|RSID=rs10401969
|Name_s=
|Gene_s=SF4
|Feature=
|Evidence=PubMed ID:19060906; Web Resource:http://www.genome.gov/gwastudies/
|Annotation=GWAS results: Common variants at 30 loci contribute to polygenic dyslipidemia. (Initial Sample Size: 19,840 individuals; Replication Sample Size: Up to 20,623 individuals); (Region: 19p13.11; Reported Gene(s): NCAN, CILP2, PBX4; Risk Allele: rs10401969-C); (p-value= 0.00000002).This variant is associated with LDL cholesterol.
|Drugs=
|Drug Classes=
|Diseases=
|Curation Level=Non-Curated
|PharmGKB Accession ID=PA164740251
}}

{{PMID Auto GWAS
|PMID=20864672
|Trait=None
|Title=Genetic Variants Influencing Circulating Lipid Levels and Risk of Coronary Artery Disease
|RiskAllele=T
|Pval=1E-11
|OR=0.05
|ORtxt=[0.04-0.06] unit increase
|OA=1
}}

{{PMID Auto GWAS
|PMID=20686565
|Trait=None
|Title=Biological, clinical and population relevance of 95 loci for blood lipids.
|RiskAllele=C
|Pval=2E-29
|OR=7.8300
|ORtxt=None
|OA=1
}}

{{PMID Auto
|PMID=19656773
|Title=A polygenic basis for four classical Fredrickson hyperlipoproteinemia phenotypes that are characterized by hypertriglyceridemia.
|OA=1
}}

{{PMID Auto
|PMID=19951432
|Title=Analysis of recently identified dyslipidemia alleles reveals two loci that contribute to risk for carotid artery disease.
|OA=1
}}

{{GET Evidence
|impact=pathogenic
|qualified_impact=Insufficiently evaluated pathogenic
|inheritance=unknown
|quality_scores=Array
|dbsnp_id=rs10401969
|n_genomes=10
|n_genomes_annotated=0
|n_haplomes=13
|n_articles=0
|n_articles_annotated=0
|in_gwas=Y
|in_pharmgkb=Y
|autoscore=2
|webscore=N
}}

{{PMID Auto
|PMID=23119086
|Title=Variants Identified in a GWAS Meta-Analysis for Blood Lipids Are Associated with the Lipid Response to Fenofibrate
|OA=1
}}

{{PMID Auto
|PMID=23092954
|Title=SHAVE: shrinkage estimator measured for multiple visits increases power in GWAS of quantitative traits.
|OA=1
}}

{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | Affy GenomeWide 6}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}