{{Rsnum
|rsid=104894504
|Chromosome=15
|Orientation=plus
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
|Gene=TPM1
|position=63057028
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|Gene_s=TPM1
}}{{omim
|id=191010
|rsnum=104894504
|variant=0003
}}{{ClinVar
|rsid=104894504
|Reversed=0
|FwdREF=T
|FwdALT=C
|REF=T
|ALT=C
|RSPOS=63349227
|CHROM=15
|dbSNPBuildID=132
|SSR=0
|SAO=1
|VP=0x050368000000000002110100
|GENEINFO=TPM1:7168
|GENE_NAME=TPM1
|GENE_ID=7168
|WGT=0
|VC=SNV
|CLNALLE=1
|CLNHGVS=NC_000015.9:g.63349227T>C
|CLNSRC=Leiden Muscular Dystrophy pages (TPM1); OMIM Allelic Variant
|CLNORIGIN=1
|CLNSRCID=TPM1_00007; 191010.0003
|CLNSIG=5
|CLNCUI=C1861863
|CLNDBN=Familial hypertrophic cardiomyopathy 3
|Disease=Familial hypertrophic cardiomyopathy 3
|CLNACC=RCV000013273.22
|Tags=PM;PMC;S3D;SLO;OTHERKG;LSD;OM
|CLNDSDB=GeneReviews:MedGen:OMIM
|CLNDSDBID=NBK1768:C1861863:115196
}}{{PMID Auto
|PMID=11136687
|Title=Hypertrophic cardiomyopathy caused by a novel alpha-tropomyosin mutation (V95A) is associated with mild cardiac phenotype, abnormal calcium binding to troponin, abnormal myosin cycling, and poor prognosis.
}}

{{PMID Auto
|PMID=12858563
|Title=Molecular epidemiology of hypertrophic cardiomyopathy.
}}