{{Rsnum
|rsid=1071583
|Gene=FCN1
|Chromosome=9
|position=134909954
|Orientation=plus
|ReferenceAllele=G
|GMAF=0.3627
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
|Gene_s=FCN1
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 38.1 | 44.2 | 17.7
| HCB | 25.7 | 52.9 | 21.3
| JPT | 22.1 | 46.9 | 31.0
| YRI | 93.2 | 6.8 | 0.0
| ASW | 68.4 | 29.8 | 1.8
| CHB | 25.7 | 52.9 | 21.3
| CHD | 25.7 | 48.6 | 25.7
| GIH | 54.5 | 42.6 | 3.0
| LWK | 90.9 | 8.2 | 0.9
| MEX | 24.6 | 29.8 | 45.6
| MKK | 87.8 | 12.2 | 0.0
| TSI | 40.0 | 47.0 | 13.0
| HapMapRevision=28
}}

{{PMID|18032536}} the A allele [[rs2989727]] was significantly increased in [[RA]] patients (67%) compared with controls (60%) (P = 0.002). Also, the frequency of the G allele of [[rs1071583]] was increased in RA patients (68%) compared with controls (61%) (P = 0.003).

{{PMID Auto
|PMID=22940091
|Title=Polymorphisms in the lectin pathway genes as a possible cause of early chronic Pseudomonas aeruginosa colonization in cystic fibrosis patients.
}}

{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}