{{Rsnum
|rsid=10761129
|Gene=ROR2
|Chromosome=9
|position=91724039
|Orientation=plus
|ReferenceAllele=G
|MissenseAllele=A
|GMAF=0.2158
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
|Gene_s=ROR2
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 15.2 | 38.4 | 46.4
| HCB | 2.2 | 21.2 | 76.6
| JPT | 0.0 | 6.3 | 93.7
| YRI | 2.0 | 34.7 | 63.3
| ASW | 7.0 | 38.6 | 54.4
| CHB | 2.2 | 21.2 | 76.6
| CHD | 0.0 | 17.4 | 82.6
| GIH | 20.8 | 40.6 | 38.6
| LWK | 1.8 | 41.3 | 56.9
| MEX | 5.2 | 25.9 | 69.0
| MKK | 2.6 | 16.7 | 80.8
| TSI | 2.9 | 46.1 | 51.0
| HapMapRevision=28
}}{{Venter SNP
|rsid=10761129
|allele=T
|frequency=0.627
|uid=1103652127483
|type=homozygous_SNP
|hugo=ROR2
|ensembl gene=ENSG00000169071
|ensembl transcript=ENST00000375708
|sift=TOLERATED
|disease=Defects in ROR2 are a cause of recessive Robinow syndrome (RRS) (MIM:268310). RRS is an autosomal disorder characterized by skeletal dysplasia with generalized limb bone shortening, segmental defects of the spine, brachydactyly and a dysmorphic facial appearance.
}}

{{GET Evidence
|gene=ROR2
|aa_change=Val819Ile
|aa_change_short=V819I
|impact=not reviewed
|qualified_impact=Insufficiently evaluated not reviewed
|inheritance=unknown
|quality_scores=Array
|dbsnp_id=rs10761129
|overall_frequency_n=7572
|overall_frequency_d=10750
|overall_frequency=0.704372
|n_genomes=50
|n_genomes_annotated=0
|n_haplomes=82
|n_articles=0
|n_articles_annotated=0
|qualityscore_in_silico=2
|qualitycomment_in_silico=Y
|gene_in_genetests=Y
|genetests_testable=Y
|genetests_reviewed=Y
|nblosum100=-4
|autoscore=3
|n_web_uneval=5
}}

{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}