{{Rsnum
|rsid=17121510
|Chromosome=11
|position=118005440
|Orientation=plus
|GMAF=0.1446
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;G)
| geno3=(G;G)
| CEU | 78.8 | 20.4 | 0.9
| HCB | 92.7 | 7.3 | 0.0
| JPT | 87.6 | 12.4 | 0.0
| YRI | 51.7 | 44.2 | 4.1
| ASW | 49.1 | 45.6 | 5.3
| CHB | 92.7 | 7.3 | 0.0
| CHD | 95.4 | 4.6 | 0.0
| GIH | 83.2 | 14.9 | 2.0
| LWK | 39.1 | 44.5 | 16.4
| MEX | 81.0 | 17.2 | 1.7
| MKK | 42.9 | 50.0 | 7.1
| TSI | 69.6 | 29.4 | 1.0
| HapMapRevision=28
}}A study of maternal and fetal DNA from 370 US Caucasian birth-events (172 cases and 198 controls) concluded that the single strongest effect in fetal DNA was associated with [[rs17121510]] in the interleukin-10 receptor antagonist (IL-10RA) gene at both allelic (p = 0.01) and genotypic (p = 3.34x10e-4) levels. The odds ratio for [[preterm birth]] for the genotypic additive model was 1.92 (CI: 1.15-3.19, p = 2.00x10e-3). {{PMID|18818748|OA=1
}}

{{PMID|20463618|OA=1
}} Role of polymorphic variants as genetic modulators of infection in neonatal sepsis.

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | Affy GenomeWide 6}}
{{on chip | FTDNA2}}
{{on chip | FTDNA}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}