{{Rsnum
|rsid=1983132
|Chromosome=4
|position=177298634
|Orientation=plus
|GMAF=0.219
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 84.7 | 14.4 | 0.9
| HCB | 78.1 | 20.4 | 1.5
| JPT | 71.7 | 25.7 | 2.7
| YRI | 20.4 | 57.1 | 22.4
| ASW | 33.3 | 52.6 | 14.0
| CHB | 78.1 | 20.4 | 1.5
| CHD | 74.8 | 24.3 | 0.9
| GIH | 61.4 | 33.7 | 5.0
| LWK | 32.7 | 50.9 | 16.4
| MEX | 65.5 | 29.3 | 5.2
| MKK | 27.6 | 53.2 | 19.2
| TSI | 74.5 | 21.6 | 3.9
| HapMapRevision=28
}}[[rs1983132]] is a SNP in the [[NEIL3]] gene on ch 4, which encodes a glycosylase that can initiate base excision repair.

A study of pesticide use and SNPs in ~800 [[prostate cancer]] cases revealed a significant association between men with either a [[rs1983132]](C;T) or (T;T) genotype exposed to the pesticide [[fonofos]] and disease risk. The odds ratios and for low and high pesticide use versus no use were 1.65 (CI:0.91-3.01) and 3.25 (CI:1.78-5.92), respectively, whereas fonofos was not associated with prostate cancer risk among men with a [[rs1983132]](C;C) genotype.{{PMID|21810555|OA=1
}}

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | FTDNA}}
{{on chip | Illumina Human 1M}}
{{on chip | NatGeo2}}