{{Rsnum
|rsid=2241524
|Gene=LILRA2
|Chromosome=19
|position=55098667
|Orientation=minus
|GMAF=0.1015
|Assembly=GRCh37
|GenomeBuild=37.1
|dbSNPBuild=131
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
}}
{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 100.0 | 0.0 | 0.0
| HCB | 50.0 | 47.7 | 2.3
| JPT | 51.2 | 44.2 | 4.7
| YRI | 93.7 | 6.3 | 0.0
| ASW | 0.0 | 0.0 | 0.0
| CHB | 50.0 | 47.7 | 2.3
| CHD | 0.0 | 0.0 | 0.0
| GIH | 0.0 | 0.0 | 0.0
| LWK | 0.0 | 0.0 | 0.0
| MEX | 0.0 | 0.0 | 0.0
| MKK | 0.0 | 0.0 | 0.0
| TSI | 0.0 | 0.0 | 0.0
| HapMapRevision=28
}}
[[rs2241524]] is a SNP in the [[LILRA2]] gene that disrupts the splice acceptor site of intron 6.

Case-control association studies on 273 Japanese patients with systemic lupus erythematosus ([[SLE]]) and 50 patients with microscopic polyangiitis (MPA) showed an association between the homozygous [[rs2241524]](T;T) genotype (as oriented in dbSNP). For the association with SLE, the odds ratio was  1.82 (CI: 1.02-3.24, p=0.041) and for MPA, the odds ratio was 2.52 (CI: 1.07-5.96, p=0.049).{{PMID|18273033}}{{PMID Auto
|PMID=17903297
|Title=Genetic correlates of brain aging on MRI and cognitive test measures: a genome-wide association and linkage analysis in the Framingham Study.
|OA=1
}}

{{PMID Auto
|PMID=20236493
|Title=The contribution of genetic variation and infection to the pathogenesis of ANCA-associated systemic vasculitis.
|OA=1
}}

{{on chip | NatGeo2}}