{{Rsnum
|rsid=2302009
|Gene=CCL26
|Chromosome=7
|position=75769680
|Orientation=minus
|GMAF=0.264
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(G;G)
|geno2=(G;T)
|geno3=(T;T)
|Gene_s=CCL26
}}{{ population diversity
| geno1=(G;G)
| geno2=(G;T)
| geno3=(T;T)
| CEU | 7.1 | 36.3 | 56.6
| HCB | 0.0 | 16.8 | 83.2
| JPT | 0.9 | 20.4 | 78.8
| YRI | 19.3 | 49.0 | 31.7
| ASW | 21.1 | 45.6 | 33.3
| CHB | 0.0 | 16.8 | 83.2
| CHD | 1.9 | 22.2 | 75.9
| GIH | 12.0 | 38.0 | 50.0
| LWK | 19.3 | 47.7 | 33.0
| MEX | 0.0 | 33.3 | 66.7
| MKK | 15.4 | 44.2 | 40.4
| TSI | 4.9 | 31.4 | 63.7
| HapMapRevision=28
}}[[rs2302009]] is a SNP located in the 3' untranslated region of the [[CCL26]] gene. This gene (and this SNP) were discovered to be strongly associated with 117 patients with [[eosinophilic esophagitis]], a form of [[esophagitis]] (or inflammation of the esophagus) that shares many symptoms with gastroesophageal reflux disease ([[GERD]]). {{PMID|16453027|OA=1
}}

[[rs2302009]](G) is the risk allele. The [[rs2302009(G;G)]] homozygous genotype is the most predisposed to developing [[eosinophilic esophagitis]] (odds ratio 4.55, CI 1.71â€“12.39). The heterozgyote ([[rs2302009(G;T)]]) genotype's risk was indistinguishable statistically from the normal [[rs23020099(T;T)]] genotype. {{PMID|15453027}}

{{omim
| id = 604697
| variant = 0001
| quiet = 1
| desc    = ESOPHAGITIS, EOSINOPHILIC, SUSCEPTIBILITY TO
| rsnum   = 2302009
}}

{{omim
|desc=ESOPHAGITIS, EOSINOPHILIC
|id=610247
|rsnum=2302009
}}

{{PMID Auto
|PMID=17848170
|Title=The chemokine network. II. On how polymorphisms and alternative splicing increase the number of molecular species and configure intricate patterns of disease susceptibility.
|OA=1
}}

{{PMID Auto
|PMID=20808897
|Title=Disease-associated mutations that alter the RNA structural ensemble.
|OA=1
}}

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}