{{Rsnum
|rsid=251124
|Gene=VCAN
|Chromosome=5
|position=83509605
|Orientation=plus
|GMAF=0.2282
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
|Gene_s=VCAN
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 64.9 | 34.2 | 0.9
| HCB | 44.9 | 42.6 | 12.5
| JPT | 51.8 | 38.4 | 9.8
| YRI | 70.8 | 26.4 | 2.8
| ASW | 62.5 | 28.6 | 8.9
| CHB | 44.9 | 42.6 | 12.5
| CHD | 32.4 | 50.0 | 17.6
| GIH | 54.5 | 39.6 | 5.9
| LWK | 70.6 | 25.7 | 3.7
| MEX | 69.0 | 29.3 | 1.7
| MKK | 51.9 | 41.7 | 6.4
| TSI | 64.4 | 31.7 | 4.0
| HapMapRevision=28
}}{{PMID|18484070}} failed to reproduce evidence of intracranial aneurysms in a Han Chinese population

[http://stroke.ahajournals.org/cgi/reprint/37/9/2372 journal] rs251124 (OR=1.43,95% CI=1.09 to 1.88, P=0.008) disruption of the extracellular matrix (ECM) of the arterial wall and is a likely factor in the pathogenesis of intracranial aneurysms (IAs).

{{PMID Auto
|PMID=19506372
|Title=Association analysis of genes involved in the maintenance of the integrity of the extracellular matrix with intracranial aneurysms in a Japanese cohort
}}

{{PMID Auto
|PMID=23568740
|Title=[Association of single nucleotide polymorphisms of CSPG2 and HSPG2 genes with intracranial aneurysm in ethnic Han Chinese population]
}}

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}