{{Rsnum
|rsid=2736654
|geno1=(A;A)
|geno2=(A;C)
|geno3=(C;C)
|Status = Merged
|Merged = 4746
|Orientation=minus
|Chromosome=6
|position=38682852
|Gene=GLO1
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|Gene_s=GLO1
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;C)
| geno3=(C;C)
| CEU | 29.2 | 44.6 | 26.2
| HCB | 77.8 | 22.2 | 0.0
| JPT | 93.2 | 6.8 | 0.0
| YRI | 54.0 | 36.5 | 9.5
| ASW | 0.0 | 0.0 | 0.0
| CHB | 77.8 | 22.2 | 0.0
| CHD | 0.0 | 0.0 | 0.0
| GIH | 0.0 | 0.0 | 0.0
| LWK | 0.0 | 0.0 | 0.0
| MEX | 0.0 | 0.0 | 0.0
| MKK | 0.0 | 0.0 | 0.0
| TSI | 0.0 | 0.0 | 0.0
| HapMapRevision=28
}}
{{PMID Auto
|PMID=19412133
|Title=Polymorphisms in glyoxalase 1 gene are not associated with vascular complications: the Hoorn and CoDAM studies
}}

{{omim
|id=138750
|desc=GLYOXALASE I; GLO1
|rsnum=2736654
}}
{{PMID Auto
|PMID=21491613
|Title=Glyoxalase I polymorphism rs2736654 causing the Ala111Glu substitution modulates enzyme activity-implications for autism
|OA=1
}}

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}