{{Rsnum
|rsid=3131713
|Gene=PINK1
|Chromosome=1
|position=20645555
|Orientation=plus
|GMAF=0.1745
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
|Gene_s=PINK1,PINK1-AS
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;G)
| geno3=(G;G)
| CEU | 76.1 | 21.2 | 2.7
| HCB | 67.2 | 29.9 | 2.9
| JPT | 54.0 | 42.5 | 3.5
| YRI | 47.6 | 44.2 | 8.2
| ASW | 54.4 | 36.8 | 8.8
| CHB | 67.2 | 29.9 | 2.9
| CHD | 60.6 | 34.9 | 4.6
| GIH | 74.3 | 23.8 | 2.0
| LWK | 60.9 | 33.6 | 5.5
| MEX | 65.5 | 32.8 | 1.7
| MKK | 65.4 | 30.1 | 4.5
| TSI | 81.4 | 18.6 | 0.0
| HapMapRevision=28
}}This SNP may be related to [[obesity]] and [[diabetes]].

Genomic variants at the [[PINK1]] locus are associated with transcript abundance and plasma nonesterified fatty acid concentrations in European whites. Near perfect linkage disequilibrium with [[rs622525]]. G allele is a proxy for [[rs622525]](T) which is associated with higher [[PINK1]] transcript levels.{{PMID|18495756}}

{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | Illumina Human 1M}}