{{Rsnum
|rsid=561241
|Gene=F7
|Chromosome=13
|position=113105720
|Orientation=minus
|GMAF=0.1028
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
|Gene_s=F7
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;G)
| geno3=(G;G)
| CEU | 81.5 | 15.4 | 3.1
| HCB | 91.1 | 8.9 | 0.0
| JPT | 88.6 | 11.4 | 0.0
| YRI | 74.6 | 25.4 | 0.0
| ASW | 0.0 | 0.0 | 0.0
| CHB | 91.1 | 8.9 | 0.0
| CHD | 0.0 | 0.0 | 0.0
| GIH | 0.0 | 0.0 | 0.0
| LWK | 0.0 | 0.0 | 0.0
| MEX | 0.0 | 0.0 | 0.0
| MKK | 0.0 | 0.0 | 0.0
| TSI | 0.0 | 0.0 | 0.0
| HapMapRevision=28
}}{{PMID|17903294|OA=1
}} risk of cardiovascular disease

[[rs561241]] for hemostatic factors was p = 4.5*10(-16)

{{PMID|17903291|OA=1
}} The Framingham Heart Study 100K SNP genome-wide association study resource: overview of 17 phenotype working group reports.

{{PMID|19474294|OA=1
}} Potential etiologic and functional implications of genome-wide association loci for human diseases and traits.

{{PMID|19822575|OA=1
}} Molecular interactions between HNF4a, FOXA2 and GABP identified at regulatory DNA elements through ChIP-sequencing.

{{PMID Auto GWAS
|PMID=22703881
|Trait=None
|Title=Genetic Associations for Activated Partial Thromboplastin Time and Prothrombin Time, their Gene Expression Profiles, and Risk of Coronary Artery Disease.
|RiskAllele=C
|Pval=4E-56
|OR=0.0350
|ORtxt=None
|OA=1
}}

{{GET Evidence
|impact=pathogenic
|qualified_impact=Insufficiently evaluated pathogenic
|inheritance=unknown
|quality_scores=Array
|dbsnp_id=rs561241
|overall_frequency_n=15
|overall_frequency_d=126
|overall_frequency=0.119048
|n_genomes=10
|n_genomes_annotated=0
|n_haplomes=11
|n_articles=0
|n_articles_annotated=0
|in_gwas=Y
|autoscore=1
|webscore=N
}}

{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | HumanOmni1Quad}}