{{Rsnum
|rsid=6837293
|Gene=PRKG2
|Chromosome=4
|position=81141777
|Orientation=plus
|GMAF=0.4642
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;T)
|geno3=(T;T)
|effect1=
|effect2=
|effect3=
|Gene_s=PRKG2
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;T)
| geno3=(T;T)
| CEU | 70.8 | 27.4 | 1.8
| HCB | 16.9 | 55.9 | 27.2
| JPT | 25.2 | 45.0 | 29.7
| YRI | 0.0 | 2.8 | 97.2
| ASW | 5.3 | 24.6 | 70.2
| CHB | 16.9 | 55.9 | 27.2
| CHD | 14.0 | 49.5 | 36.4
| GIH | 42.6 | 44.6 | 12.9
| LWK | 0.0 | 5.6 | 94.4
| MEX | 39.3 | 50.0 | 10.7
| MKK | 2.6 | 26.3 | 71.2
| TSI | 55.4 | 40.6 | 4.0
| HapMapRevision=28
}}

Two SNPs in the cGMP-dependent protein kinase II (cGK II) [[PRKG2]] gene, [[rs7688672]] and [[rs6837293]], were found to be associated in 148 matched Taiwanese case:control subjects with [[gout]] disease using a recessive model after adjustment of hyperuricemia (odds ratio 2.72, CI: 1.13-6.54).{{PMID|18678579}}

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}