{{Rsnum
|rsid=7482144
|Gene=HBG2
|Chromosome=11
|position=5254939
|Orientation=plus
|GMAF=0.1685
|Gene_s=HBG2,LOC100288908
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
}}{{omim
|desc=SICKLE CELL ANEMIA
|id=603903
|rsnum=7482144
}}

{{omim
|desc=FETAL HEMOGLOBIN QUANTITATIVE TRAIT LOCUS 1; HBFQTL1
|id=141749
|rsnum=7482144
}}

{{omim
|id=142250
|desc=HEMOGLOBIN, GAMMA G; HBG2
|rsnum=7482144
}}

{{PMID Auto
|PMID=20472475
|Title=The XmnI (G)gamma polymorphism influences hemoglobin F synthesis contrary to BCL11A and HBS1L-MYB SNPs in a cohort of 57 beta-thalassemia intermedia patients
}}

{{omim
|id=142250
|rsnum=7482144
|variant=0028
}}

{{PMID|18194558|OA=1
}} A hierarchical and modular approach to the discovery of robust associations in genome-wide association studies from pooled DNA samples.

{{PMID|18667698|OA=1
}} DNA polymorphisms at the BCL11A, HBS1L-MYB, and beta-globin loci associate with fetal hemoglobin levels and pain crises in sickle cell disease.

{{PMID|18695233|OA=1
}} Genetic complexity in sickle cell disease.

{{PMID|19148297|OA=1
}} Genetic variation on chromosome 6 influences F cell levels in healthy individuals of African descent and HbF levels in sickle cell patients.

{{PMID|20353593|OA=1
}} Genetic modifiers of Hb E/beta0 thalassemia identified by a two-stage genome-wide association study.

{{PMID|20401335|OA=1
}} Sickle Cell Disease in the Post Genomic Era: A Monogenic Disease with a Polygenic Phenotype.

{{on chip | 23andMe v3}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}