{{Rsnum
|rsid=7688672
|Gene=PRKG2
|Chromosome=4
|position=81148295
|Orientation=plus
|GMAF=0.4605
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
|effect1=
|effect2=
|effect3=
|Gene_s=PRKG2
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;G)
| geno3=(G;G)
| CEU | 1.5 | 30.8 | 67.7
| HCB | 28.1 | 54.8 | 17.0
| JPT | 29.7 | 43.2 | 27.0
| YRI | 94.6 | 4.8 | 0.7
| ASW | 71.4 | 23.2 | 5.4
| CHB | 28.1 | 54.8 | 17.0
| CHD | 37.7 | 50.0 | 12.3
| GIH | 13.1 | 45.5 | 41.4
| LWK | 90.9 | 9.1 | 0.0
| MEX | 12.1 | 50.0 | 37.9
| MKK | 66.0 | 32.1 | 1.9
| TSI | 4.0 | 41.0 | 55.0
| HapMapRevision=28
}}

Two SNPs in the cGMP-dependent protein kinase II (cGK II) [[PRKG2]] gene, [[rs7688672]] and [[rs6837293]], were found to be associated in 148 matched Taiwanese case:control subjects with [[gout]] disease using a recessive model after adjustment of hyperuricemia (odds ratio 2.72, CI: 1.13-6.54).{{PMID|18678579}}

{{on chip | 23andMe v1}}
{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | FTDNA2}}
{{on chip | HumanOmni1Quad}}
{{on chip | Illumina Human 1M}}