{{Rsnum
|rsid=8702
|Gene=KLC1
|Chromosome=14
|position=103686015
|Orientation=minus
|GMAF=0.4431
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(C;C)
|geno2=(C;G)
|geno3=(G;G)
|Gene_s=KLC1
}}{{ population diversity
| geno1=(C;C)
| geno2=(C;G)
| geno3=(G;G)
| CEU | 52.4 | 38.1 | 9.5
| HCB | 11.1 | 57.8 | 31.1
| JPT | 31.0 | 40.5 | 28.6
| YRI | 21.0 | 58.1 | 21.0
| ASW | 0.0 | 0.0 | 0.0
| CHB | 11.1 | 57.8 | 31.1
| CHD | 0.0 | 0.0 | 0.0
| GIH | 0.0 | 0.0 | 0.0
| LWK | 0.0 | 0.0 | 0.0
| MEX | 0.0 | 0.0 | 0.0
| MKK | 0.0 | 0.0 | 0.0
| TSI | 0.0 | 0.0 | 0.0
| HapMapRevision=28
}}
SNP [[rs8702]], located in the [[KLC1]] gene, has been reported to have a protective effect against the occurrence of [[multiple sclerosis]]. Specifically, the odds of a [[rs8702(G;G)]] individual - as oriented to the dbSNP entry, not as published - are reported to be 0.21 (CI: 0.018 - 0.88). {{PMID|17999208}}

A study of ~800 Swedish patients with [[Alzheimer's disease]] did not find an association to [[rs8702 ]] independently, however, [[rs8702]] interacted with [[ApoE4]] carrier status in AD (p=0.006).{{PMID|17611642}}

{{PMID Auto
|PMID=19911314
|Title=Kinesin Light Chain 1 Gene Haplotypes in Three Conformational Diseases
}}

{{PMID|17653041|OA=1
}} Variability in the kinesin light chain 1 gene may influence risk of age-related cataract.

{{PMID|19046961}} Gene-environmental effects behind leukoaraiosis: a silent genetic variant of the kinesin protein can be activated in a subject with poorly controlled long-lasting hypertension.

{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | HumanOmni1Quad}}