{{Rsnum
|rsid=873978
|Gene=FAAH
|Chromosome=1
|position=46408231
|Orientation=minus
|GMAF=0.1157
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
|Gene_s=FAAH
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;G)
| geno3=(G;G)
| CEU | 0.0 | 0.9 | 99.1
| HCB | 8.8 | 42.3 | 48.9
| JPT | 14.2 | 37.2 | 48.7
| YRI | 3.4 | 19.0 | 77.6
| ASW | 0.0 | 24.6 | 75.4
| CHB | 8.8 | 42.3 | 48.9
| CHD | 8.3 | 43.1 | 48.6
| GIH | 5.9 | 37.6 | 56.4
| LWK | 0.0 | 16.5 | 83.5
| MEX | 0.0 | 19.0 | 81.0
| MKK | 0.6 | 13.5 | 85.9
| TSI | 0.0 | 4.9 | 95.1
| HapMapRevision=28
}}{{PMID Auto
|PMID=20044928
|Title=Mutation screen and association studies for the fatty acid amide hydrolase (FAAH) gene and early onset and adult obesity
|OA=1
}}

{{PMID|19014633|OA=1
}} Lack of association of genetic variants in genes of the endocannabinoid system with anorexia nervosa.

{{PMID|20033240|OA=1
}} Eating disorders: the current status of molecular genetic research.

{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | Affy GenomeWide 6}}