{{Rsnum
|rsid=894160
|Gene=PLIN1
|Chromosome=15
|position=89668592
|Orientation=minus
|GMAF=0.3324
|Assembly=GRCh38
|GenomeBuild=38.1
|dbSNPBuild=141
|geno1=(A;A)
|geno2=(A;G)
|geno3=(G;G)
|Gene_s=PLIN1
}}{{ population diversity
| geno1=(A;A)
| geno2=(A;G)
| geno3=(G;G)
| CEU | 10.7 | 42.9 | 46.4
| HCB | 20.4 | 44.5 | 35.0
| JPT | 12.5 | 46.4 | 41.1
| YRI | 8.8 | 44.9 | 46.3
| ASW | 15.8 | 36.8 | 47.4
| CHB | 20.4 | 44.5 | 35.0
| CHD | 15.6 | 50.5 | 33.9
| GIH | 4.0 | 41.6 | 54.5
| LWK | 10.9 | 39.1 | 50.0
| MEX | 15.5 | 39.7 | 44.8
| MKK | 3.2 | 37.2 | 59.6
| TSI | 7.8 | 43.1 | 49.0
| HapMapRevision=28
}}"We studied a population-based sample of Caribbean-origin Hispanics (n = 920, aged 45-74 y) living in the Boston area. Obesity measures (waist and hip circumference, BMI) did not differ between GG subjects and carriers of the A allele (GA and AA). In multivariate linear regression models, we found a significant interaction between complex carbohydrate intake as a continuous variable and PLIN 11482 G > A genotype for waist circumference (P = 0.002). By dichotomizing complex carbohydrate intake, we found significantly different effects across PLIN 11482G > A genotypes. When complex carbohydrate intake was <144 g/d, waist circumference was larger in PLIN 11482G > A carriers (P = 0.024). Conversely, when complex carbohydrate intake was >/=144 g/d, waist and hip circumferences were less in PLIN 11482G > A carriers (P < 0.05). These interactions were not found for simple sugars or total carbohydrates. In subjects with higher complex carbohydrate intake, the minor allele was protective against [[obesity]], whereas in subjects with lower carbohydrate intake, the minor allele was associated with increased obesity." {{PMID|18806092|OA=1
}}

{{PMID Auto
|PMID=19399648
|Title=Possible role for ENPP1 polymorphism in obesity but not for INSIG2 and PLIN variants
}}

{{PMID|16836753|OA=1
}} Study of the impact of perilipin polymorphisms in a French population.

{{PMID|18356850|OA=1
}} Common variations in perilipin gene, central obesity, and risk of type 2 diabetes in US women.

{{PMID|18812483|OA=1
}} Effects of perilipin (PLIN) gene variation on metabolic syndrome risk and weight loss in obese children and adolescents.

{{PMID|19375517|OA=1
}} PAT proteins, an ancient family of lipid droplet proteins that regulate cellular lipid stores.

{{PMID|19385027}} Relationship between perilipin gene polymorphisms and body weight and body composition during weight loss and weight maintenance.

{{PMID|21193293|OA=1
}} Perilipin polymorphism interacts with saturated fat and carbohydrates to modulate insulin resistance.

{{PMID|21392418}} Preliminary findings on the role of PLIN1 polymorphisms on body composition and energy metabolism response to energy restriction in obese women.

{{PMID Auto
|PMID=23111648
|Title=Evaluation of the ENPP1 and PLIN Single Nucleotide Polymorphisms With Type 2 Diabetes in a Taiwanese Population:  Evidence for Replication and Gene-Gene Interaction
}}

{{PMID Auto
|PMID=23517113
|Title=Association between three genetic variants of the Perilipin Gene (PLIN) and glucose metabolism: results from a replication study among Chinese adults and a meta-analysis
}}

{{on chip | 23andMe v2}}
{{on chip | 23andMe v3}}
{{on chip | 23andMe v4}}
{{on chip | Affy GenomeWide 6}}